A common mechanism for microcephaly

New studies employing high-throughput parallel sequencing have revealed _WDR62_ mutations in individuals with microcephaly associated with a broad range of malformations of cortical

development. These findings establish that WDR62 acts as a molecular link between proliferation and migration in neurogenesis. Access through your institution Buy or subscribe This is a

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during checkout ADDITIONAL ACCESS OPTIONS: * Log in * Learn about institutional subscriptions * Read our FAQs * Contact customer support REFERENCES * Thornton, G.K. et al. _Trends Genet._

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Article  CAS  Google Scholar  * Bilgüvar, K. et al. _Nature_ 467, 207–210 (2010). Article  Google Scholar  Download references AUTHOR INFORMATION AUTHORS AND AFFILIATIONS * Bernd Wollnik is

at the Center for Molecular Medicine Cologne (CMMC), Institute of Human Genetics, and Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of

Cologne, Cologne, Germany., Bernd Wollnik Authors * Bernd Wollnik View author publications You can also search for this author inPubMed Google Scholar CORRESPONDING AUTHOR Correspondence to

Bernd Wollnik. ETHICS DECLARATIONS COMPETING INTERESTS The author declares no competing financial interests. RIGHTS AND PERMISSIONS Reprints and permissions ABOUT THIS ARTICLE CITE THIS

ARTICLE Wollnik, B. A common mechanism for microcephaly. _Nat Genet_ 42, 923–924 (2010). https://doi.org/10.1038/ng1110-923 Download citation * Published: 27 October 2010 * Issue Date:

November 2010 * DOI: https://doi.org/10.1038/ng1110-923 SHARE THIS ARTICLE Anyone you share the following link with will be able to read this content: Get shareable link Sorry, a shareable

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