A common mechanism for microcephaly

New studies employing high-throughput parallel sequencing have revealed _WDR62_ mutations in individuals with microcephaly associated with a broad range of malformations of cortical

development. These findings establish that WDR62 acts as a molecular link between proliferation and migration in neurogenesis. Access through your institution Buy or subscribe This is a

preview of subscription content, access via your institution RELEVANT ARTICLES Open Access articles citing this article. * A DE NOVO VARIANT IN ADGRL2 SUGGESTS A NOVEL MECHANISM UNDERLYING

THE PREVIOUSLY UNDESCRIBED ASSOCIATION OF EXTREME MICROCEPHALY WITH SEVERELY REDUCED SULCATION AND RHOMBENCEPHALOSYNAPSIS * Myriam Vezain * , Matthieu Lecuyer *  … Pascale Saugier-Veber

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* Log in * Learn about institutional subscriptions * Read our FAQs * Contact customer support REFERENCES * Thornton, G.K. et al. _Trends Genet._ 25, 501–510 (2009). Article  CAS  Google

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et al. _Nature_ 467, 207–210 (2010). Article  Google Scholar  Download references AUTHOR INFORMATION AUTHORS AND AFFILIATIONS * Bernd Wollnik is at the Center for Molecular Medicine Cologne

(CMMC), Institute of Human Genetics, and Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of Cologne, Cologne, Germany., Bernd Wollnik Authors

* Bernd Wollnik View author publications You can also search for this author inPubMed Google Scholar CORRESPONDING AUTHOR Correspondence to Bernd Wollnik. ETHICS DECLARATIONS COMPETING

INTERESTS The author declares no competing financial interests. RIGHTS AND PERMISSIONS Reprints and permissions ABOUT THIS ARTICLE CITE THIS ARTICLE Wollnik, B. A common mechanism for

microcephaly. _Nat Genet_ 42, 923–924 (2010). https://doi.org/10.1038/ng1110-923 Download citation * Published: 27 October 2010 * Issue Date: November 2010 * DOI:

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