Reactive oxygen species and the segregation of mtDNA sequence variants
Reactive oxygen species and the segregation of mtDNA sequence variants"
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Mutations in mitochondrial DNA (mtDNA) are an important cause of a group of human diseases collectively referred to as mitochondrial encephalomyopathies. Most pathogenic mtDNA mutations
coexist with wild-type genomes, and an oxidative phosphorylation defect is not manifest until the proportion of mutant genomes exceeds a particular cellular threshold. The pattern of
segregation of mtDNA mutations is thus a key determinant of the onset and severity of disease. The mechanisms regulating tissue-specific mtDNA segregation, however, remain largely unknown.
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Reactive oxygen species and the segregation of mtDNA sequence variantsMutations in mitochondrial DNA (mtDNA) are an important cause of a group of human diseases collectively referred to as m...
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